Labels:text | screenshot | black and white | black | font | document OCR: on cases Very occasionally (roughly one in a mi or the LDL an individual inherits a abnormal gene receptor from both parents, leading to a mia in homozygous familial hypercholesterola LDI which there are essentially no function h very receptors. This situation is associated w high levels of LDL and very accelerated atherosclerotic complications, nich also Recently, a genetic mutation of apo B, w he results in defective removal of LDL via 1. The LDL-receptor pathway, has been describe mewhat frequency of familial defective apo B is The less than familial hypercholesterolaemi picture to condition results in an identical clinica that seen in heterozygous familial hypercholesterolaemia.
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